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Analytic Morphomics in Myositis-Related Interstitial Lung Disease.

dc.contributor.authorO'Mahony, Alexander T
dc.contributor.authorHenry, Patrick J
dc.contributor.authorCoghlan, Patrick
dc.contributor.authorWaldron, Michael
dc.contributor.authorCrowley, Claire
dc.contributor.authorRyan, David
dc.contributor.authorMoore, Niamh
dc.contributor.authorBennett, Deirdre M
dc.contributor.authorO'Connor, Owen J
dc.contributor.authorMaher, Michael M
dc.contributor.authorHenry, Michael T
dc.date.accessioned2024-12-18T15:28:20Z
dc.date.available2024-12-18T15:28:20Z
dc.date.issued2023-07-17
dc.identifier.pmid37458801
dc.identifier.doi10.1007/s00408-023-00637-3
dc.identifier.urihttp://hdl.handle.net/10147/643793
dc.descriptionFVC and DLCO decreased but within acceptable limits of treatment response (FVC: 83.7-78.7%, p < 0.05, DLCO 63.4-60.6%, p < 0.05). The cross-sectional area of the PM and ESM increased (PM: 39.8 to 40.7 cm2, p = 0.491; ESM: 35.2 to 39.5 cm2, p = 0.098). Density significantly fell for both the PM and ESM (PM: 35.3-31 HU, p < 0.05; ESM: 38-33.7, p < 0.05). Subcutaneous fat area increased from 103.9 to 136.1 cm2 (p < 0.05), while the visceral fat area increased but not reaching statistical significance. The change in PM density between time points demonstrated an inverse correlation with DLCO (p < 0.05, R =  - 0.49).en_US
dc.language.isoenen_US
dc.rights© 2023. The Author(s).
dc.rightsAttribution 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/*
dc.subjectAnalytic morphomicsen_US
dc.subjectbody compositionen_US
dc.subjectIdiopathic inflammatory myopathiesen_US
dc.subjectInterstitial lung diseaseen_US
dc.titleAnalytic Morphomics in Myositis-Related Interstitial Lung Disease.en_US
dc.typeArticleen_US
dc.typeOtheren_US
dc.identifier.eissn1432-1750
dc.identifier.journalLungen_US
dc.source.journaltitleLung
dc.source.volume201
dc.source.issue4
dc.source.beginpage345
dc.source.endpage353
refterms.dateFOA2024-12-18T15:28:22Z
dc.source.countryUnited States


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