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dc.contributor.authorLyons, Paul A
dc.contributor.authorRayner, Tim F
dc.contributor.authorTrivedi, Sapna
dc.contributor.authorHolle, Julia U
dc.contributor.authorWatts, Richard A
dc.contributor.authorJayne, David R W
dc.contributor.authorBaslund, Bo
dc.contributor.authorBrenchley, Paul
dc.contributor.authorBruchfeld, Annette
dc.contributor.authorChaudhry, Afzal N
dc.contributor.authorCohen Tervaert, Jan Willem
dc.contributor.authorDeloukas, Panos
dc.contributor.authorFeighery, Conleth
dc.contributor.authorGross, Wolfgang L
dc.contributor.authorGuillevin, Loic
dc.contributor.authorGunnarsson, Iva
dc.contributor.authorHarper, Lorraine
dc.contributor.authorHrušková, Zdenka
dc.contributor.authorLittle, Mark A
dc.contributor.authorMartorana, Davide
dc.contributor.authorNeumann, Thomas
dc.contributor.authorOhlsson, Sophie
dc.contributor.authorPadmanabhan, Sandosh
dc.contributor.authorPusey, Charles D
dc.contributor.authorSalama, Alan D
dc.contributor.authorSanders, Jan-Stephan F
dc.contributor.authorSavage, Caroline O
dc.contributor.authorSegelmark, Mårten
dc.contributor.authorStegeman, Coen A
dc.contributor.authorTesař, Vladimir
dc.contributor.authorVaglio, Augusto
dc.contributor.authorWieczorek, Stefan
dc.contributor.authorWilde, Benjamin
dc.contributor.authorZwerina, Jochen
dc.contributor.authorRees, Andrew J
dc.contributor.authorClayton, David G
dc.contributor.authorSmith, Kenneth G C
dc.date.accessioned2012-11-28T15:29:04Z
dc.date.available2012-11-28T15:29:04Z
dc.date.issued2012-07-19
dc.identifier.citationGenetically distinct subsets within ANCA-associated vasculitis. 2012, 367 (3):214-23 N. Engl. J. Med.en_GB
dc.identifier.issn1533-4406
dc.identifier.pmid22808956
dc.identifier.doi10.1056/NEJMoa1108735
dc.identifier.urihttp://hdl.handle.net/10147/253772
dc.description.abstractAntineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a severe condition encompassing two major syndromes: granulomatosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis. Its cause is unknown, and there is debate about whether it is a single disease entity and what role ANCA plays in its pathogenesis. We investigated its genetic basis.
dc.language.isoenen
dc.publisherThe New England journal of medicineen_GB
dc.rightsArchived with thanks to The New England journal of medicineen_GB
dc.subject.meshAnti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis
dc.subject.meshCase-Control Studies
dc.subject.meshFemale
dc.subject.meshGenetic Predisposition to Disease
dc.subject.meshGenome-Wide Association Study
dc.subject.meshGenotyping Techniques
dc.subject.meshHLA-DP Antigens
dc.subject.meshHumans
dc.subject.meshMajor Histocompatibility Complex
dc.subject.meshMale
dc.subject.meshMicroscopic Polyangiitis
dc.subject.meshMyeloblastin
dc.subject.meshPolymorphism, Single Nucleotide
dc.subject.meshRisk Factors
dc.subject.meshWegener Granulomatosis
dc.subject.meshalpha 1-Antitrypsin
dc.titleGenetically distinct subsets within ANCA-associated vasculitis.en_GB
dc.typeArticle In Pressen
dc.contributor.departmentCambridge Institute for Medical Research, and Department of Medicine, University of Cambridge School of Clinical Medicine, Addenbrooke's Hospital, Cambridge, United Kingdom. kgcs2@cam.ac.uken_GB
dc.identifier.journalThe New England journal of medicineen_GB
dc.description.provinceLeinsteren
html.description.abstractAntineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a severe condition encompassing two major syndromes: granulomatosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis. Its cause is unknown, and there is debate about whether it is a single disease entity and what role ANCA plays in its pathogenesis. We investigated its genetic basis.


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