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    Genetically distinct subsets within ANCA-associated vasculitis.

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    Authors
    Lyons, Paul A
    Rayner, Tim F
    Trivedi, Sapna
    Holle, Julia U
    Watts, Richard A
    Jayne, David R W
    Baslund, Bo
    Brenchley, Paul
    Bruchfeld, Annette
    Chaudhry, Afzal N
    Cohen Tervaert, Jan Willem
    Deloukas, Panos
    Feighery, Conleth
    Gross, Wolfgang L
    Guillevin, Loic
    Gunnarsson, Iva
    Harper, Lorraine
    Hrušková, Zdenka
    Little, Mark A
    Martorana, Davide
    Neumann, Thomas
    Ohlsson, Sophie
    Padmanabhan, Sandosh
    Pusey, Charles D
    Salama, Alan D
    Sanders, Jan-Stephan F
    Savage, Caroline O
    Segelmark, Mårten
    Stegeman, Coen A
    Tesař, Vladimir
    Vaglio, Augusto
    Wieczorek, Stefan
    Wilde, Benjamin
    Zwerina, Jochen
    Rees, Andrew J
    Clayton, David G
    Smith, Kenneth G C
    Show allShow less
    Affiliation
    Cambridge Institute for Medical Research, and Department of Medicine, University of Cambridge School of Clinical Medicine, Addenbrooke's Hospital, Cambridge, United Kingdom. kgcs2@cam.ac.uk
    Issue Date
    2012-07-19
    MeSH
    Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis
    Case-Control Studies
    Female
    Genetic Predisposition to Disease
    Genome-Wide Association Study
    Genotyping Techniques
    HLA-DP Antigens
    Humans
    Major Histocompatibility Complex
    Male
    Microscopic Polyangiitis
    Myeloblastin
    Polymorphism, Single Nucleotide
    Risk Factors
    Wegener Granulomatosis
    alpha 1-Antitrypsin
    Show allShow less
    
    Metadata
    Show full item record
    Citation
    Genetically distinct subsets within ANCA-associated vasculitis. 2012, 367 (3):214-23 N. Engl. J. Med.
    Publisher
    The New England journal of medicine
    Journal
    The New England journal of medicine
    URI
    http://hdl.handle.net/10147/253772
    DOI
    10.1056/NEJMoa1108735
    PubMed ID
    22808956
    Abstract
    Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a severe condition encompassing two major syndromes: granulomatosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis. Its cause is unknown, and there is debate about whether it is a single disease entity and what role ANCA plays in its pathogenesis. We investigated its genetic basis.
    Item Type
    Article In Press
    Language
    en
    ISSN
    1533-4406
    ae974a485f413a2113503eed53cd6c53
    10.1056/NEJMoa1108735
    Scopus Count
    Collections
    Tallaght University Hospital

    entitlement

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