The role of thromboxane A(2) in the pathogenesis of intrauterine growth restriction associated with maternal smoking in pregnancy.
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Affiliation
Coombe Women and Infants University Hospital, Dublin, Ireland., caoimhemlynch@eircom.netIssue Date
2012-02-01T10:57:10ZMeSH
6-Ketoprostaglandin F1 alpha/analogs & derivatives/urineAdult
Case-Control Studies
Female
Fetal Growth Retardation/*etiology/metabolism
Humans
Pregnancy
Smoking/*adverse effects
Thromboxane A2/*metabolism
Thromboxane B2/analogs & derivatives/urine
Young Adult
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Prostaglandins Other Lipid Mediat. 2011 Aug;95(1-4):63-7. doi:, 10.1016/j.prostaglandins.2011.06.007. Epub 2011 Jun 23.Journal
Prostaglandins & other lipid mediatorsDOI
10.1016/j.prostaglandins.2011.06.007PubMed ID
21723954Abstract
BACKGROUND: To examine the effect of maternal smoking in pregnancy on the production of two eicosanoids, thromboxane A(2) and prostacyclin I2, and their role in the pathogenesis of intrauterine growth restriction. METHODS: Prospective case control study enrolled smoking and non-smoking women atLanguage
engISSN
1098-8823 (Print)1098-8823 (Linking)
ae974a485f413a2113503eed53cd6c53
10.1016/j.prostaglandins.2011.06.007
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