The role of thromboxane A(2) in the pathogenesis of intrauterine growth restriction associated with maternal smoking in pregnancy.

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Authors
Lynch, Caoimhe M
O'Kelly, Ruth
Stuart, Bernard
Treumann, Achim
Conroy, Ronan
Regan, Carmen L
Affiliation
Coombe Women and Infants University Hospital, Dublin, Ireland., caoimhemlynch@eircom.net
Issue Date
2012-02-01T10:57:10Z
MeSH
6-Ketoprostaglandin F1 alpha/analogs & derivatives/urine
Adult
Case-Control Studies
Female
Fetal Growth Retardation/*etiology/metabolism
Humans
Pregnancy
Smoking/*adverse effects
Thromboxane A2/*metabolism
Thromboxane B2/analogs & derivatives/urine

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Citation
Prostaglandins Other Lipid Mediat. 2011 Aug;95(1-4):63-7. doi:, 10.1016/j.prostaglandins.2011.06.007. Epub 2011 Jun 23.
Journal
Prostaglandins & other lipid mediators
URI
http://hdl.handle.net/10147/207996
DOI
10.1016/j.prostaglandins.2011.06.007
PubMed ID
21723954
Abstract
BACKGROUND: To examine the effect of maternal smoking in pregnancy on the production of two eicosanoids, thromboxane A(2) and prostacyclin I2, and their role in the pathogenesis of intrauterine growth restriction. METHODS: Prospective case control study enrolled smoking and non-smoking women at
Language
eng
ISSN
1098-8823 (Print)
1098-8823 (Linking)
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