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    The role of thromboxane A(2) in the pathogenesis of intrauterine growth restriction associated with maternal smoking in pregnancy.

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    Authors
    Lynch, Caoimhe M
    O'Kelly, Ruth
    Stuart, Bernard
    Treumann, Achim
    Conroy, Ronan
    Regan, Carmen L
    Affiliation
    Coombe Women and Infants University Hospital, Dublin, Ireland., caoimhemlynch@eircom.net
    Issue Date
    2012-02-01T10:57:10Z
    MeSH
    6-Ketoprostaglandin F1 alpha/analogs & derivatives/urine
    Adult
    Case-Control Studies
    Female
    Fetal Growth Retardation/*etiology/metabolism
    Humans
    Pregnancy
    Smoking/*adverse effects
    Thromboxane A2/*metabolism
    Thromboxane B2/analogs & derivatives/urine
    Young Adult
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    Citation
    Prostaglandins Other Lipid Mediat. 2011 Aug;95(1-4):63-7. doi:, 10.1016/j.prostaglandins.2011.06.007. Epub 2011 Jun 23.
    Journal
    Prostaglandins & other lipid mediators
    URI
    http://hdl.handle.net/10147/207996
    DOI
    10.1016/j.prostaglandins.2011.06.007
    PubMed ID
    21723954
    Abstract
    BACKGROUND: To examine the effect of maternal smoking in pregnancy on the production of two eicosanoids, thromboxane A(2) and prostacyclin I2, and their role in the pathogenesis of intrauterine growth restriction. METHODS: Prospective case control study enrolled smoking and non-smoking women at
    Language
    eng
    ISSN
    1098-8823 (Print)
    1098-8823 (Linking)
    ae974a485f413a2113503eed53cd6c53
    10.1016/j.prostaglandins.2011.06.007
    Scopus Count
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    Coombe Women & Infants University Hospital

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