The role of thromboxane A(2) in the pathogenesis of intrauterine growth restriction associated with maternal smoking in pregnancy.
AffiliationCoombe Women and Infants University Hospital, Dublin, Ireland., firstname.lastname@example.org
MeSH6-Ketoprostaglandin F1 alpha/analogs & derivatives/urine
Fetal Growth Retardation/*etiology/metabolism
Thromboxane B2/analogs & derivatives/urine
MetadataShow full item record
CitationProstaglandins Other Lipid Mediat. 2011 Aug;95(1-4):63-7. doi:, 10.1016/j.prostaglandins.2011.06.007. Epub 2011 Jun 23.
JournalProstaglandins & other lipid mediators
AbstractBACKGROUND: To examine the effect of maternal smoking in pregnancy on the production of two eicosanoids, thromboxane A(2) and prostacyclin I2, and their role in the pathogenesis of intrauterine growth restriction. METHODS: Prospective case control study enrolled smoking and non-smoking women at
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