Hdl Handle:
http://hdl.handle.net/10147/232975
Title:
Cardiac biomarkers in neonatal hypoxic ischaemia.
Authors:
Sweetman, D; Armstrong, K; Murphy, J F A; Molloy, E J
Affiliation:
Neonatology, National Maternity Hospital, Dublin, Ireland. dsweetman@nmh.ie
Citation:
Cardiac biomarkers in neonatal hypoxic ischaemia. 2012, 101 (4):338-43 Acta Paediatr.
Journal:
Acta paediatrica (Oslo, Norway : 1992)
Issue Date:
Apr-2012
URI:
http://hdl.handle.net/10147/232975
DOI:
10.1111/j.1651-2227.2011.02539.x
PubMed ID:
22118561
Abstract:
Following a perinatal hypoxic-ischaemic insult, term infants commonly develop cardiovascular dysfunction. Troponin-T, troponin-I and brain natriuretic peptide are sensitive indicators of myocardial compromise. The long-term effects of cardiovascular dysfunction on neurodevelopmental outcome following perinatal hypoxic ischaemia remain controversial. Follow-up studies are warranted to ensure optimal cardiac function in adulthood. CONCLUSION: Cardiac biomarkers may improve the diagnosis of myocardial injury, help guide management, estimate mortality risk and may also aid in longterm neurodevelopmental outcome prediction following neonatal hypoxic-ischaemia.
Item Type:
Article
Language:
en
MeSH:
Biological Markers; Electrocardiography; Humans; Hypoxia-Ischemia, Brain; Infant, Newborn; Myocardial Ischemia; Natriuretic Peptide, Brain; Troponin I; Troponin T
ISSN:
1651-2227

Full metadata record

DC FieldValue Language
dc.contributor.authorSweetman, Den_GB
dc.contributor.authorArmstrong, Ken_GB
dc.contributor.authorMurphy, J F Aen_GB
dc.contributor.authorMolloy, E Jen_GB
dc.date.accessioned2012-07-10T14:07:07Z-
dc.date.available2012-07-10T14:07:07Z-
dc.date.issued2012-04-
dc.identifier.citationCardiac biomarkers in neonatal hypoxic ischaemia. 2012, 101 (4):338-43 Acta Paediatr.en_GB
dc.identifier.issn1651-2227-
dc.identifier.pmid22118561-
dc.identifier.doi10.1111/j.1651-2227.2011.02539.x-
dc.identifier.urihttp://hdl.handle.net/10147/232975-
dc.description.abstractFollowing a perinatal hypoxic-ischaemic insult, term infants commonly develop cardiovascular dysfunction. Troponin-T, troponin-I and brain natriuretic peptide are sensitive indicators of myocardial compromise. The long-term effects of cardiovascular dysfunction on neurodevelopmental outcome following perinatal hypoxic ischaemia remain controversial. Follow-up studies are warranted to ensure optimal cardiac function in adulthood. CONCLUSION: Cardiac biomarkers may improve the diagnosis of myocardial injury, help guide management, estimate mortality risk and may also aid in longterm neurodevelopmental outcome prediction following neonatal hypoxic-ischaemia.en_GB
dc.language.isoenen
dc.rightsArchived with thanks to Acta paediatrica (Oslo, Norway : 1992)en_GB
dc.subject.meshBiological Markers-
dc.subject.meshElectrocardiography-
dc.subject.meshHumans-
dc.subject.meshHypoxia-Ischemia, Brain-
dc.subject.meshInfant, Newborn-
dc.subject.meshMyocardial Ischemia-
dc.subject.meshNatriuretic Peptide, Brain-
dc.subject.meshTroponin I-
dc.subject.meshTroponin T-
dc.titleCardiac biomarkers in neonatal hypoxic ischaemia.en_GB
dc.typeArticleen
dc.contributor.departmentNeonatology, National Maternity Hospital, Dublin, Ireland. dsweetman@nmh.ieen_GB
dc.identifier.journalActa paediatrica (Oslo, Norway : 1992)en_GB
dc.description.provinceLeinsteren

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